coagulative

Coagulative necrosis of the hepatocytes can occur around the central vein.

Dry gangrene is actually a form of coagulative necrosis.

New technique for liver resection using heat coagulative necrosis.

In coagulative necrosis the architecture of dead tissue is preserved for at least a couple of days.

Coagulative necrosis occurs primarily in tissues such the kidney, heart and adrenal glands.

Coagulative necrosis is a type of accidental cell death typically caused by ischemia or infarction.

Coagulative necrosis occurs here.

Coagulative tumor cell necrosis is common in clinically malignant smooth muscle cell tumors.

Under the microscope, myocardial infarction presents as a circumscribed area of ischemic, coagulative necrosis (cell death).

Wet gangrene is coagulative necrosis progressing to liquefactive necrosis.

One exception to coagulative necrosis is the brain, which undergoes liquefactive necrosis in response to infarction.

Coagulative necrosis is most commonly caused by conditions that do not involve severe trauma, toxins or an acute or chronic immune response.

The energy delivered with each sonication is capable of raising the temperature of the tissue upwards of 65 degrees Celsius resulting in coagulative necrosis.

In electrosurgical devices, high bursts of energy are used for short periods of time to achieve cutting and coagulative effects on tissues [ 12 13 14 ] .

Liquefactive necrosis (or colliquative necrosis), in contrast to coagulative necrosis, is characterized by the digestion of dead cells to form a viscous liquid mass.

Caseous necrosis can be considered a combination of coagulative and liquefactive necroses, typically caused by mycobacteria (e.g. tuberculosis), fungi and some foreign substances.

At the focus of this ultrasound energy, the temperature can reach excesses of 80 C which results in nearly spontaneous coagulative necrosis or cell death without harming neighboring cells.

The cone-shaped ultrasound beam penetrates through soft tissue and produces well defined regions of protein denaturation, irreversible cell damage, and coagulative necrosis, at specific target locations.

The macroscopic appearance of an area of coagulative necrosis is a pale segment of tissue contrasting against surrounding well vascularised tissue and is dry on cut surface.

Grossly, the tumors are usually hemorrhagic and soft and microscopically marked by pleomorphism, abundant (15-30 per 10 high power fields) abnormal mitotic figures, and coagulative tumor cell necrosis.

Coagulative necrosis is characterized by the formation of a gelatinous (gel-like) substance in dead tissues in which the architecture of the tissue is maintained, and can be observed by light microscopy.

These central regions begin to die through coagulative necrosis, though they also retain some of the structural characteristics of previously normal tissues, enabling a distinction from the granulomas of tuberculosis where caseous necrosis obliterates preexisting structures.

It is important to note that while ischemia in most tissues of the body will cause coagulative necrosis, in the central nervous system ischemia causes liquefactive necrosis as there is very little structural framework in neural tissue.

A specific type of phytobezoar, termed a diospyrobezoar, is associated with ingestion of unripe persimmons, which contain a soluble tannin called shibuol that polymerizes into a coagulative cellulose-protein compound in the acid environment of the stomach, to form the bezoar.

When the hilar lymph node for instance is infected with tuberculosis and leads to caseous necrosis, its gross appearance can be a cheesy tan to white, which is why this type of necrosis is often depicted as a combination of both coagulative and liquefactive necrosis.