These analyses offer an explanation for the findings that there are no common mutations shared by most cancers.

A rival theory to explain common mutations is that they just happened to be present in a founding population and grew more frequent by chance.

Prenatal tests can tell whether their unborn child has the most common mutations.

Theories regarding the biochemical significance of the two most common mutations are noted below:

Overexposure to ultraviolet radiation induces at least two common genetic mutations.

The most common mutations, R1141X and 23-29del, account for about 25% of the found mutations.

The most common mutation is the deamination of cytosine to uracil.

U6a and U6b share a common basal mutation (16219) that is not present in U6c.

Haplogroups U9 and U4 share two common mutations at the root of their phylogeny.

On the other hand, I don't have plenty of common nasty mutations, such as those associated with colon cancer and melanoma.