The nematode Caenorhabditis elegans has a condensin I-related complex involved in dosage compensation (DCC).

X inactivation plays a key role in dosage compensation.

The nematode Caenorhabditis elegans has an SMC4-variant that has a specialized role in dosage compensation.

Another example is acetylation of H4K16, which has been associated with dosage compensation of the male X chromosome in Drosophila melanogaster.

To correct this imbalance, mammalian females have evolved a unique mechanism of dosage compensation.

We suggest that trisomy of other chromosomes may exceed a limit of survivable dosage compensation during development.

This protein is normally expressed in males, where it promotes male development and prevents dosage compensation.

Species can have different mechanisms of dosage compensation.

An exception to this phenomenon of dosage compensation is the level of steroid sulfatase in blood, which is increased in women compared with men.

He later demonstrated that there were multiple genes on the Drosophila Y chromosome, and described the mechanism of dosage compensation.