Notably, frog embryos figured prominently in a study that used evolutionary principles to identify a novel vascular disrupting agent that may have chemotherapeutic potential.

With a hot needle, he killed one of the two cells of the frog embryo after the first cleavage and then watched the development of the surviving cell.

This is just another example of regulation but it also shows that in the early frog embryo the fate of the parts is not fixed.

An old and elegant result, at first puzzling, involves combining the surface sheet of cells of a frog embryo with the underlying tissues from near the mouth region of the newt.

In frog embryos, cyclin E-Cdk2 is primarily responsible for activating origins.

Genes that make a jellyfish glow in the dark were implanted in frog embryos, and the large eyes of the resulting tadpoles shine in the dark like green traffic lights.

The experiments used frog embryos that were incubated with 1:5000 dilutions of a commercial glyphosate solution.

Thomas King, whose early research in frog embryos foreshadowed the cloning of sheep and other mammals decades later, died last Wednesday at Johns Hopkins Hospital in Baltimore.

Tiabendazole serves to block angiogenesis in both frog embryos and human cells.

A cDNA library from the blastula stage of a frog embryo was cloned into RNA expression plasmids to generate synthetic mRNA.