Several thousand human diseases are caused by one or more defective or missing genes of the 50,000 to 100,000 genes in every human cell.

The researchers then mixed the stem-progenitor cell mix with a virus that contained the missing gene.

The missing genes are from a chromosome contributed by the child's father.

Many of the missing genes are encoded in the nuclear genome of the host.

The missing gene is known as nef, and its function is not fully known.

Thalassemia is caused by variant or missing genes that affect how the body makes hemoglobin.

In order to propagate such viral vectors, a cell line that expresses the missing genes is required.

When the scientists reinserted the missing gene in the laboratory, the bacterium once again became vulnerable to the drug.

Cones that gained the missing genes began expressing the new photopigment.

Scientists removed white blood cells from the patient, grew them in the lab, and inserted the missing gene into the cells.