Its effects derive from the fact that it binds to and activates mu opioid receptors in the brain, spinal cord, stomach and intestine.

Morphine binds to and activates mu opioid receptor in the brain, spinal cord, stomach and intestine.

Both analgesia and drug addiction are functions of the mu opioid receptor, the class of opioid receptor first identified as responsive to morphine.

Azidomorphine binds with high affinity to the mu opioid receptor, and is around 40x more potent than morphine in vivo.

It has also been shown to produce analgesic effects, most probably acting as a partial agonist at the mu opioid receptor.

Despite its anesthetic and analgesic effects, etoxadrol does not interact with benzodiazepine, muscarinic acetylcholine, or mu opioid receptors.

The opioid processes involve mu opioid receptors and are present in the rostromedial shell part of the nucleus accumbens on its spiny neurons.

Mice lacking the mu opioid receptor exhibited a lack of conditioned place preference.

This analgesic effect has been studied in animals and shown to be reversible by naloxone, suggesting a mu opioid receptor mediated effect.

It is an agonist at mu opioid receptors.