Keeping in mind that the great predominance of rat genes have direct counterparts in humans, learning more about the genetic base of their behavior will be most enlightening.

Expression of the rat gene is induced by peroxisome proliferators.

HBII-52 refers to the human gene, whereas RBII-52 is used for the rat gene and MBII-52 is used for naming the mouse gene.

A short segment of identity was found in exon 1a with exon 2 of the mouse, human, and rat genes, suggesting it is orthologous to these exons, and thus should lie upstream from exon 1b (see table 1 - see additional file 1).

This product was not further investigated since it is not a human or rat gene.

Putative initiation and stop codons for the human nestin gene were found at the same positions as in the rat gene, in regions where overall similarity was very high.

RGD curators review the literature and manually curate GO, disease, phenotype and pathways for rat genes, disease and pathways for mouse genes, and disease, phenotype and pathways for human genes.

The expression of the rat gene is upregulated by retinoic acid, which results in a specific reduction in EAAC1-mediated glutamate transport.

The homologous region of the rat gene lacks the Ets-1-like NF binding site, but instead binds another NF expressed in rat thymocytes but not splenocytes.

Dr. Lima and Dr. Miesenböck added genetic on-switches to the rat gene so that it would become active only in a particular type of neuron in a fly.