Light and repetitive visual signals can induce epileptic seizures (see Bucha effect).

The excitatory signals induce contraction of the muscle cells, which causes the joint angles in the arm to change, pulling the arm away.

These signals also induce the synthesis of crystallins.

In the presence of CRMP-2, the signal can induce alterations of Rac-dependent pathway, which modulates the actin filament assembly in the growth cone.

Several pro-apoptotic signals, such as TNF, TRAIL, and FADD, also induce autophagy.

This signal will ultimately induce a release of several substances among which are dopamine and norepinephrine, resulting in an antinociceptive effect on the organism.

Frequently, extracellular signals induce the expression of immediate early genes (IEGs) such as c-fos, c-jun, or AP-1.

Upon stimulation, a cellular signal induces a conformational change in the integrin cytoplasmic domain that propagates to the extracellular domain.

A signal from MS at the 12-cell stage induces pharynx in ABa progeny cells but not in ABp progeny.

Light and repetitive visual signals can induce epileptic seizures.