Infection occurs rapidly, with the virus adhering to surface receptors within 15 minutes of entering the respiratory tract.

These might include a 30kDa surface receptor, or macrophage scavenger receptors.

Rudd was the first to discover that intracellular protein kinases can interact with surface receptors.

This may include the regulation of several genes, the production of other cytokines, or an increase in surface receptors for other molecules.

Local protein synthesis is required to maintain the number of surface receptors at the synapse.

Protective antigen binds to two surface receptors on the host cell.

The invasion process requires a coupling of the actin-myosin motor to the surface receptors.

These surface receptors allow a bridge to be formed which binds to host endothelial cells.

Food lectins can also interact with the surface receptors of the body's white cells, programming them to multiply rapidly.

This set of protein complexes is critical for down-regulating activated cell surface receptors.