Thiazides are useful for treating absorptive hypercalciuria, a condition in which high urinary calcium is a result of excess absorption from the gastrointestinal tract.

Caffeine, urinary calcium, calcium metabolism and bone.

Increased urinary calcium is an expected compensatory response with associated increased risk of renal calculi.

Sustained reduction in urinary calcium during long-term treatment with slow release neutral potassium phosphate in absorptive hypercalciuria.

Thiazides also lower urinary calcium excretion, making them useful in preventing calcium-containing kidney stones.

The increase in urinary calcium can also contribute to kidney stones.

High dietary protein: Research has found an association between diets high in animal protein and increased urinary calcium, and have been linked to an increase in fractures.

A clinical study with Phyllanthus niruri, indicated that it may reduce the levels of urinary calcium.

In a urinalysis, the normal amount of urinary calcium can be measured in amount per time (commonly per 24 hours).

An abnormally high amount of urinary calcium is called hypercalciuria and an abnormally low amount is called hypocalcuria.